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It's formulated to address the biochemical root of why high-achievers can't switch off and to build the kind of nervous system resilience that holds, long term

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the control only reached 35 % When they blocked VEGFR2 (with the selective inhibitor SU5416), all of these effects disappeared Why it matters: The last point is what holds the study together

The use of ICR mice as a preclinical model continues to provide critical insights, bridging the gap between basic research and clinical applications in AD therapy